Data CitationsChan L. KC, Seizl M, Tresch A, Cramer P. 2012. Cramer (2) mRNA halflife. Electron Microscopy Data Bank. E-MTAB-760Wang Y, Liu CL, Storey JD, Tibshirani RJ, Herschlag D, Brown PO. 2002. Brown (1) and (2) mRNA halflife. Stanford Genomic Resources. Yuleidatafiles/halflifeTable.txtDuttagupta R, Tian B, Wilusz CJ, Khounh DT, Soteropoulos P, Ouyang M, Dougherty JP, Peltz SW. MS-275 inhibitor database 2005. Peltz mRNA halflife, Supplementary Tables S1, S2, and S3. PubMed Central. molcellb_25_13_5499__index.htmlPresnyak V, Alhusaini N, Chen YH, Martin S, Morris N, Kline N, Olson S, Weinberg D, Baker KE, Graveley BR, Coller J. 2015. Coller (1) and (2) mRNA halflife, Table S1. PubMed Central. NIHMS665436-supplement-5.xlsGrigull J, Mnaimneh WBP4 S, Pootoolal J, Robinson MD, Hughes TR. 2004. Hughes mRNA stability. Hughes Lab, University of Toronto. Grigull/Fig2A_data.xlsHolstege FC, Jennings EG, Wyrick JJ, Lee TI, Hengartner CJ, Green MR, Golub TR, Lander ES, Young RA. 1998. Young mRNA stability. Young Lab, Whitehead Institute for Biomedical Research. young/expression.htmlGeisberg JV. 2014. Struhl mRNA stability, Table S2. PubMed Central. NIHMS552811-supplement-2.xlsxShalem O, Dahan O, Levo M, Martinez MR, Furman I, Segal E, Pilpel Y. 2008. Pipel mRNA stability, Supplementary Table 1. PubMed Central. msb200859-s2.xlsMunchel SE, Shultzaberger RK, Takizawa N. 2011. Weis (1) mRNA stability, Supplemental Data. Molecular Biology of the Cell. mc-e11-01-0028-s10.xlsPelechano V, Prez-Ortn JE. 2010. Perez-Ortin mRNA stability. Wiley Online Library. yea_1768_supportinforTS1.xlsSupplementary MaterialsFigure 1source data 1: Source data for Figure 1B: decay kinetics of ACT1, CIS3 and RPL25 mRNAs. elife-32536-fig1-data1.txt (220K) DOI:?10.7554/eLife.32536.005 Figure 1source data 2: Source data for Figure 1C and Figure 1figure supplement 1C: transcriptome wide decay data for two biological replicates of wild-type yeast cells grown in exponential phase. elife-32536-fig1-data2.txt (220K) DOI:?10.7554/eLife.32536.006 Figure 1figure supplement 2source data 1: Source MS-275 inhibitor database data for Shape 1figure health supplement 2ACB: transcriptome wide decay data ready without polyA selection. elife-32536-fig1-figsupp2-data1.txt (121K) DOI:?10.7554/eLife.32536.007 Figure 3source data 1: Resource data for Figure 3B, C, DCG, I and J: decay kinetics of selected mRNAs in cells treated with translational inhibitors. elife-32536-fig3-data1.xlsx (53K) DOI:?10.7554/eLife.32536.014 Figure 3source data 2: Resource data for Figure 3H: Transcriptome wide decay data for cells treated or mock treated with 3AT. elife-32536-fig3-data2.txt (132K) DOI:?10.7554/eLife.32536.015 Figure 3source data 3: Resource data for Figure 3K: Transcriptome wide decay data for cells treated with DMSO, hippuristanol or cycloheximide. elife-32536-fig3-data3.txt (162K) DOI:?10.7554/eLife.32536.016 Shape 4source data 1: Resource data for Shape 4B, C and F: accumulation kinetics of decay and P-bodies of RNA in P-bodies in cells treated with translational inhibitors. elife-32536-fig4-data1.xlsx (44K) DOI:?10.7554/eLife.32536.021 Supplementary file 1: Resources of MS-275 inhibitor database data for Shape 2. elife-32536-supp1.docx (16K) DOI:?10.7554/eLife.32536.022 Supplementary document 2: Candida strains. elife-32536-supp2.docx (17K) DOI:?10.7554/eLife.32536.023 Supplementary file 3: Plasmids. elife-32536-supp3.docx (15K) DOI:?10.7554/eLife.32536.024 Supplementary file 4: qPCR primers. elife-32536-supp4.docx (14K) DOI:?10.7554/eLife.32536.025 Supplementary file 5: Plasmid sequences. elife-32536-supp5.fa (176K) DOI:?10.7554/eLife.32536.026 Transparent reporting form. elife-32536-transrepform.pdf (319K) DOI:?10.7554/eLife.32536.027 Data Availability StatementSequencing data have already been deposited in GEO under accession code “type”:”entrez-geo”,”attrs”:”text message”:”GSE119560″,”term_identification”:”119560″GSE119560. The next dataset was generated: Chan L. 2018. mRNA balance mainly because measured by thiouracil incorporation in the absence and existence of translational inhibitors. NCBI Gene Manifestation Omnibus. GSE119560 The next previously released datasets were utilized: Nagalakshmi U, Wang Z, Waern K, Shou C, Raha D. 2008. mRNA great quantity and transcript limitations. PubMed Central. NIHMS229938-supplement-supp__dining tables.xls Subtelny AO, Eichhorn SW. 2014. polyA tail size. NCBI Gene Manifestation Omnibus. GSE52809 Brar GA, Yassour M, Friedman N, Regev A, Ingolia NT, Weissman JS. 2012. translational effectiveness. NCBI Gene Manifestation Omnibus. GSE34082 Drummond DA, Raval A, Wilke CO. 2006. CAI. Molecular Evolution and Biology. en-cai-fop.txt Pechmann S, Frydman J. 2013. nTE. Frydman Laboratory. codons/normalizedTE.txt Neymotin B, Athanasiadou R, Gresham D. 2014. Gresham mRNA halflife, Supplemental desk 5. PubMed Central. supp_20_10_1645__index.miller C html, Schwalb B, Maier K, Schulz D. 2011. Cramer (1) mRNA halflife. PubMed Central. msb2010112-s1.txt Sunlight M, Schwalb.
Tag Archives: MS-275 inhibitor database
Categories
- 34
- 5- Receptors
- A2A Receptors
- ACE
- Acetylcholinesterase
- Adenosine Deaminase
- Adenylyl Cyclase
- Adrenergic ??2 Receptors
- Alpha2 Adrenergic Receptors
- Annexin
- Antibiotics
- ATPase
- AXOR12 Receptor
- Ca2+ Ionophore
- Cannabinoid
- Cannabinoid (GPR55) Receptors
- CB2 Receptors
- CCK Receptors
- Cell Metabolism
- Cell Signaling
- Cholecystokinin2 Receptors
- CK1
- Corticotropin-Releasing Factor1 Receptors
- DHCR
- DMTases
- DNA Ligases
- DNA Methyltransferases
- Dopamine D1 Receptors
- Dopamine D3 Receptors
- Dopamine D4 Receptors
- Endothelin Receptors
- EP1-4 Receptors
- Epigenetics
- Exocytosis & Endocytosis
- Fatty Acid Synthase
- Flt Receptors
- GABAB Receptors
- GIP Receptor
- Glutamate (Kainate) Receptors
- Glutamate (Metabotropic) Group III Receptors
- Glutamate (NMDA) Receptors
- Glutamate Carboxypeptidase II
- Glycogen Phosphorylase
- Glycosyltransferase
- GnRH Receptors
- Heat Shock Protein 90
- hERG Channels
- Hormone-sensitive Lipase
- IKK
- Imidazoline Receptors
- IMPase
- Inositol Phosphatases
- Kisspeptin Receptor
- LTA4 Hydrolase
- M1 Receptors
- Matrixins
- Melastatin Receptors
- mGlu Group III Receptors
- mGlu5 Receptors
- Monoamine Oxidase
- Motilin Receptor
- My Blog
- Neutrophil Elastase
- Nicotinic (??4??2) Receptors
- NKCC Cotransporter
- NMU Receptors
- Nociceptin Receptors
- Non-Selective
- Non-selective 5-HT
- OP3 Receptors
- Opioid, ??-
- Orexin2 Receptors
- Other
- Other Oxygenases/Oxidases
- Other Transcription Factors
- p38 MAPK
- p53
- p56lck
- PAF Receptors
- PDPK1
- PKC
- PLA
- PPAR
- PPAR??
- Proteasome
- PTH Receptors
- Ras
- RNA Polymerase
- Serotonin (5-HT2B) Receptors
- Serotonin Transporters
- Sigma2 Receptors
- Sodium Channels
- Steroid Hormone Receptors
- Tachykinin NK1 Receptors
- Tachykinin NK2 Receptors
- Tachykinin, Non-Selective
- Telomerase
- Thyrotropin-Releasing Hormone Receptors
- Topoisomerase
- trpp
- Uncategorized
- USP
Recent Posts
- 2012) using the Phenotypic Characteristic Search for human strains with markers for resistance to Adamantane, Oseltamivir, or both drugs
- Tissue were homogenized into single-cell suspensions and put through red bloodstream cell lysis
- A phase I/II study investigated the safety and efficacy of concurrent local palliative RT and durvalumab (PD-L1 inhibitor) in 10 patients with unresectable or metastatic advanced solid tumors [136]
- We believe that this hypothesis-generating study could open new avenues for exploring oxidative stress as a potential pathogenetic and, hypothetically, therapeutic target for mitigating CLL strong class=”kwd-title” Keywords: Leukemia, Lymphocytic, Gilbert’s, Syndrome Gilbert’s syndrome (GS) is the most common inherited disorder of bilirubin glucuronidation
- Such costs aren’t simple for tertiary-care hospitals in growing countries sometimes, since these already are powered by minimal budget which switches into provision of fundamental medical services mostly, laboratory, radiology, pharmacy services, and bed space
Tags
a 67 kDa type I transmembrane glycoprotein present on myeloid progenitors
and differentiation. The protein kinase family is one of the largest families of proteins in eukaryotes
Apoptosis
bladder
brain
breast
cell cycle progression
cervix
CSP-B
Cyproterone acetate
EGFR) is the prototype member of the type 1 receptor tyrosine kinases. EGFR overexpression in tumors indicates poor prognosis and is observed in tumors of the head and neck
EM9
endometrium
erythrocytes
F3
Goat polyclonal to IgG H+L)
Goat polyclonal to IgG H+L)Biotin)
GRK4
GSK1904529A
Igf1
Mapkap1
monocytes andgranulocytes. CD33 is absent on lymphocytes
Mouse monoclonal to CD33.CT65 reacts with CD33 andtigen
Palomid 529
platelets
PTK) or serine/threonine
Rabbit Polyclonal to ARNT.
Rabbit polyclonal to BMPR2
Rabbit Polyclonal to CCBP2.
Rabbit Polyclonal to EDG4
Rabbit polyclonal to EIF4E.
Rabbit polyclonal to IL11RA
Rabbit polyclonal to LRRIQ3
Rabbit Polyclonal to MCM3 phospho-Thr722)
Rabbit Polyclonal to RBM34
SB 216763
SKI-606
SNX-5422
STK) kinase catalytic domains. Epidermal Growth factor receptor
stomach
stomach and in squamous cell carcinoma.
TNFSF8
TSHR
VEGFA
vulva